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Gene Replacement in the Prevention of Hereditary Mitochondrial Diseases: Three Parent Babies and Ethics

1.

İzmir Katip Çelebi Üniversitesi Sağlık Bilimleri Fakültesi Doğum ve Kadın Hastalıkları Hemşireliği Anabilim Dalı, İzmir, Türkiye

2.

Kastamonu Üniversitesi Fazıl Boyner Sağlık Bilimleri Fakültesi Ebelik Bölümü, Kastamonu, Türkiye

3.

Hacettepe Üniversitesi Hemşirelik Fakültesi Doğum ve Kadın Hastalıkları Hemşireliği Anabilim Dalı, Ankara, Türkiye

Arch Health Sci Res 2020; 7: 201-206
DOI: 10.5152/ArcHealthSciRes.2020.589394
Read: 2819 Downloads: 844 Published: 07 July 2020

Mitochondrial diseases increase mortality and morbidity caused by mutations in mitochondrial DNA (mtDNA) or functional gene disorders of mitochondria. Hereditary mutations in mtDNA are an important cause of genetic diseases for which there is no effective treatment and are transmitted from generation to generation. Mutations in DNA have an important role in infertility or other reproductive anomalies that occur later in life by affecting oocyte functions as well as negatively affecting vital organs. With the development of technology, one of the new treatment approaches developed for mitochondrial diseases is mitochondrial gene replacement therapy (MRT). MRT allows women who are carriers of mtDNA mutations to have a genetically healthy child. In this review, MRT, its effects, nursing dimensions, and ethical relationship are discussed.

Cite this article as: Evrenol Öçal S, Şimşek Çetinkaya Ş, Yücel Ç, Koç G. Gene Replacement in the Prevention of Hereditary Mitochondrial Diseases: Three Parent Babies and Ethics. Arc Health Sci Res 2020; 7(2): 201-6.

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